ABSTRACT
Objective To investigate the correlation between onset frequencies and duration of symptoms and early stroke risk in patients with transient ischemic attack (TIA).Methods From January 2013 to July 2017,inpatients with TIA at the Department of Neurology,Lu'an People's Hospital were enrolled retrospectively.According to the findings of head CT or MRI reexaminations within 72 h after the first TIA,the patients were divided into non-stroke group and stroke group.The demographic and clinical data of both groups were compared.Results A total of 171 patients with TIA were enrolled,including 54 in the stroke group and 117 in the non-stroke group.There were significant differences in sex,atrial fibrillation,using anticoagulant drug before onset,baseline blood pressure (systolic and diastolic blood pressure),lowdensity lipoprotein cholesterol,high-density lipoprotein cholesterol,fasting blood glucose,TIA etiology (cardiogenic embolism),TIA frequency (≥2 times),duration of TIA symptoms,ABCD2 score (6 and 7),and using antihypertensive drugs after onset in both groups.Multivariable logistic regression analysis indicated that high diastolic blood pressure (odds ratio [OR] 1.121,95％ confidence interval [CI] 1.016-1.238;P =0.023),high fasting glucose (OR 10.866,95％ CI 3.658-30.945;P =0.001),ABCD2 score 6 (OR 4.221,95％ CI 1.906-9.352;P ＜ 0.001),ABCD2 score 7 (OR 4.207,95％ CI 2.040-9.961,P ＜ 0.001),duration of symptoms ≥ 60 min(OR 3.682,95 ％ CI 1.961-9.989;P ＜ 0.001),and frequency of TIA onset ≥2 (OR 4.220,95％ CI 1.953-9.118;P＜0.001) were the independent risk factors for early TIA,and the use of anticoagulants before onset was an independent protective factor (OR 0.300,95％ CI 0.097-0.923;P =0.036).Conclusion TIA patients with recurrent onset (≥2 times) and duration of symptoms longer than 60 min are more likely to develop ischemic stroke in a short term.
ABSTRACT
As a key aspect of quality of life in patients w ith stroke, the clinical research of sexual function and sexual satisfaction is stil rare. The rehabilitation goals in patients w ith stroke usualy include recovering functional activities and improving quality of life, how ever, it is not yet know n about the recovery of sexual function. This article review s the epidemiology, etiology, risk factors, clinical manifestations, treatment and rehabilitation of sexual dysfunction in male patients w ith stroke.
ABSTRACT
Objective The aim of this study was to investigate the prevalence of erectile dysfunction (ED) and to assess the risk factors of ED in male post‐stroke patients .Methods One hundred and twenty six male post‐stroke patients were invited to par‐ticipate in this study .Patients were divided into ED group and non‐ED group according to the erectile function evaluated by Interna‐tional Index of Erectile Function‐5 (IIEF‐5) .The neurological impairment was measured using the National Institute of Health Stroke Scale (NIHSS) and the modified Rankin Scale (mRS) .Depressive symptoms was measured using the Hamilton Depression Scales (HAMD) .The global cognitive function was measured using the Mini mental Status Examination (MMSE) .The clinical and psychosocial factors were compared between the ED and non‐ED groups .Univariate and multivariate Logistic regression analyses were employed to assess the risk factors with ED .Results There were 38 patients (30 .2% ) with ED in all the 126 male post‐stroke patients .Univariate Logistic regression analyses revealed that hypertension ,diabetes ,ACEI ,and depression were significantly related to ED in male post‐stroke patients (P<0 .05) .Multivariate Logistic regression analyses revealed that ACEI and depression were independently associated with ED in male post‐stroke patients (P<0 .05) .Conclusion ED is common in Chinese male post‐stroke patients .ACEI and depression are the major determinants of ED .
ABSTRACT
Postprandial hyperglycemia is a risk factor for atherosclerosis and cerebro-vascular diseases. This article reviews the mechanisms of cerebrovascular diseases caused by postprandial hyperglycemia from aspects of postprandial hyperglycemia participating in the pathophysiological mechanisms of atherosclerosis, and the relations between postprandial hyperglycemia and cerebrovascular diseases.